Journal Articles
Vijayaraghavan, Tarika, Dhananjay, Samiksha, Ho, Xue Yan, Giordano-Santini, Rosina, Hilliard, Massimo and Neumann, Brent (2023). The dynamin GTPase mediates regenerative axonal fusion in Caenorhabditis elegans by regulating fusogen levels. PNAS Nexus, 2 (5) pgad114. doi: 10.1093/pnasnexus/pgad114
Ho, Xue Yan, Coakley, Sean, Amor, Rumelo, Anggono, Victor and Hilliard, Massimo A. (2022). The metalloprotease ADM-4/ADAM17 promotes axonal repair. Science Advances, 8 (11) eabm2882, eabm2882. doi: 10.1126/sciadv.abm2882
Linton, Casey, Riyadh, M. Asrafuzzaman, Ho, Xue Yan, Neumann, Brent, Giordano-Santini, Rosina and Hilliard, Massimo A. (2019). Disruption of RAB-5 increases EFF-1 fusogen availability at the cell surface and promotes the regenerative axonal fusion capacity of the neuron. Journal of Neuroscience, 39 (15), 2823-2836. doi: 10.1523/jneurosci.1952-18.2019
Lee, Irene Chengjie, Ho, Xue Yan, George, Simi Elizabeth, Goh, Catherine Wenhui, Sundaram, Jeyapriya Rajameenakshi, Pang, Karen Ka Lam, Luo, Weiwei, Yusoff, Permeen, Sze, Newman Siu Kwan and Shenolikar, Shirish (2018). Oxidative stress promotes SIRT1 recruitment to the GADD34/PP1 alpha complex to activate its deacetylase function. Cell Death and Differentiation, 25 (2), 255-267. doi: 10.1038/cdd.2017.152
Offenburger, Sarah-Lena, Ho, Xue Yan, Tachie-Menson, Theresa, Coakley, Sean, Hilliard, Massimo A. and Gartner, Anton (2018). 6-OHDA-induced dopaminergic neurodegeneration in Caenorhabditis elegans is promoted by the engulfment pathway and inhibited by the transthyretin-related protein TTR-33. PLoS Genetics, 14 (1) e1007125, 1-27. doi: 10.1371/journal.pgen.1007125
Conference Paper
Ho, X.Y., Coakley, S. and Hilliard, M.A. (2019). The metalloprotease ADM-4 promotes regenerative axonal fusion. International Worm Meeting, UCLA, September. Rockville, MD, United States: Genetics Society of America.
Thesis
Ho, Xue Yan (2020). Understanding the molecular and cellular mechanisms of axonal repair using C. elegans as a model system. PhD Thesis, Queensland Brain Institute, The University of Queensland. doi: 10.14264/b6f06f6